Cerebrovascular thrombosis (CVT) occurs in over 1,500,000 individuals yearly in the USA; of these, 66% suffer death or severe permanent paralysis.  In those suffering CVT, including transient cerebral ischemic attacks (TIA's), small stroke syndrome (SSS) and frank thrombotic stroke, at least 30% harbor a blood coagulation protein or platelet defect causing thrombosis.  

Like the disorders discussed above, the need for defining the presence or absence and type of defect is of obvious importance.  Once the defect is defined, effective therapy can be offered to significantly decrease the chances for another event.   Also, since 50% of these defects are hereditary, appropriate family members can be assessed, treated when appropriate, and potentially be saved the first thrombotic stroke or TIA.

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